An incisional hernia is a type of ventral hernia that develops at or near the site of a previous surgical incision in the abdominal wall, where abdominal contents such as intestine, omentum, or other tissue protrude through a weakened fascial defect.[1] This condition arises from inadequate healing of the surgical wound and is one of the most common long-term complications of abdominal surgery, occurring in 2–20% of patients following laparotomy procedures as of 2023.[1][2] It can remain asymptomatic or cause significant morbidity if complications develop.[1]Epidemiologically, incisional hernias affect individuals of all ages, genders, and ethnicities, with higher incidence rates observed in emergency surgeries and those involving midline incisions.[1]
Introduction
Definition
An incisional hernia is defined as an abdominal wall hernia that develops at the site of a previous surgical incision, representing a subtype of ventral hernia. This condition arises when there is a gap or defect in the fascia of the abdominal wall, allowing intra-abdominal contents to protrude through the weakened area.[1] It is specifically characterized by the presence of a bulge, with or without a palpable fascial defect, detectable via clinical examination or imaging at the postoperative scar site.[3]The hallmark presentation involves the protrusion of peritoneal contents, such as omentum or bowel, forming a visible or palpable bulge under the skin, often exacerbated by activities that increase intra-abdominal pressure. Unlike congenital hernias, which stem from innate structural weaknesses, or other acquired hernias like inguinal types that occur at natural anatomical sites, incisional hernias are distinctly iatrogenic, resulting directly from surgical disruption of the abdominal wall integrity rather than unrelated tissue vulnerabilities.[4]Incisional hernias were first systematically recognized and described in medical literature during the late 19th and early 20th centuries as a frequent complication of laparotomy procedures, coinciding with the rise of abdominal surgery. This historical acknowledgment highlighted the condition's link to postoperative wound healing failures, marking a shift from anecdotal reports to formal study in surgical outcomes.[5] Their formation is often associated with elevated intra-abdominal pressure, which strains the repaired fascia.[1]
Epidemiology
Incisional hernias occur in approximately 10-20% of patients following open abdominal surgeries, such as laparotomies, primarily due to impaired wound healing processes.[6] This rate aligns with broader estimates of up to 20% occurrence across abdominal surgeries, reflecting the challenge of fascial closure and postoperative recovery.[7] Incidence varies by incision type, with midline incisions showing rates of 3-20%, though advancements in surgical techniques, including small-bite closure methods introduced in the early 2000s and refined thereafter, have aimed to reduce these figures through improved suture practices and prophylactic mesh use.[8][9][10]Rates are notably higher in emergency procedures compared to elective ones, reaching up to 30% in urgent laparotomies due to factors like tissue trauma and contamination.[11] For instance, studies report 17.5% incidence at one year post-emergency midline laparotomy, underscoring the elevated risk in acute settings.[12] Overall, the condition manifests in 12.8-30% of cases following laparotomy, with most developing within the first few years postoperatively.[11] Recent guidelines, such as those from the European Hernia Society in 2025, continue to emphasize prevention strategies to address this burden.[13]Demographically, incisional hernias affect individuals across all ages, genders, and ethnicities, though they are more prevalent in adults who have undergone major abdominal surgery.[14] No significant disparities by race or ethnicity have been consistently identified in formation rates, but presentation and management can vary by socioeconomic factors and access to care.[15] Globally, with millions of abdominal surgeries performed annually, the condition likely results in millions of cases worldwide each year, imposing a substantial healthcare burden.[16]Obesity, a key patient-related risk factor, further exacerbates incidence in affected populations.[17]
Etiology and Risk Factors
Surgical Factors
Surgical factors play a critical role in the development of incisional hernias, primarily through intraoperative decisions and immediate postoperative complications that compromise wound integrity. Improper wound closure techniques, such as inadequate fascial approximation or the use of rapidly absorbing sutures, significantly increase the risk by failing to provide sufficient tensile strength during the healing phase. For instance, fast-absorbable sutures have been shown to elevate the incidence of incisional hernias following laparoscopic colorectal surgery compared to slowly absorbable alternatives.[18] Achieving a suture-to-woundlengthratio of at least 4:1 during closure can mitigate this risk by distributing tension more evenly across the fascia.[1]The type of incision also influences hernia formation, with midline laparotomies carrying a higher risk than transverse incisions due to greater disruption of muscle fibers and increased intra-abdominal pressure on the closure site. Studies indicate that the incidence of incisional hernia is significantly higher after midline incisions compared to transverse ones, with overall rates following laparotomy ranging from 15% to 20%.[19][1]Surgical emergencies further predispose patients to incisional hernias by introducing conditions like inflammation or contamination that heighten wound tension and impair healing. Emergency procedures are associated with elevated hernia rates owing to these acute stressors on the surgical site.[1]Wound infections and dehiscence represent direct postoperative surgical complications that create fascial gaps, facilitating hernia protrusion; in contaminated cases, these substantially contribute to long-term hernia development.[1]Technical errors, including excessive tension during closure or poor tissue handling, exacerbate these risks by promoting acute dehiscence or delayed wound healing, thereby weakening the abdominal wall early in the recovery period.[1] These surgical elements can interact with patient factors like obesity to amplify intra-abdominal pressure on the incision, heightening overall vulnerability.[1]
Patient-Related Factors
Patient-related factors play a significant role in the development of incisional hernias following abdominal surgery, primarily by compromising wound healing, increasing intra-abdominal pressure, or altering tissue integrity. These intrinsic characteristics are often non-modifiable or require preoperative optimization to mitigate risk.Obesity, defined as a body mass index (BMI) greater than 30 kg/m², is a major risk factor for incisional hernia formation, as it elevates intra-abdominal pressure and impairs wound healing through adipose tissue accumulation and chronic inflammation. Studies have reported an odds ratio of 4.74 (95% CI 1.42-15.55) for hernia development in obese patients compared to non-obese individuals, highlighting a substantially heightened vulnerability. Recurrence rates post-repair are also higher, at 13% for BMI >30 versus 8% for BMI <30.[20][21]Chronic conditions such as diabetes mellitus, chronic obstructive pulmonary disease (COPD), and malnutrition contribute to poor collagen synthesis and reduced wound strength, thereby predisposing patients to incisional hernias. Diabetes impairs glycemic control and microvascular perfusion, leading to delayed fascial repair; it has been associated with increased recurrence risk after laparoscopic repair, particularly in combination with female gender.[22] COPD elevates intra-abdominal pressure through chronic coughing and is linked to a hazard ratio of 2.35 (95% CI 1.16-4.75) for hernia development. Malnutrition depletes essential nutrients for tissue remodeling, further weakening the abdominal wall.[20][21]Smoking and corticosteroid use are modifiable patient factors that delay fascial repair by reducing vascularity and collagen deposition. Current smokers face a fourfold increased risk of incisional hernia, with an odds ratio of 3.93 (95% CI 1.82-8.49), independent of other variables, due to nicotine-induced vasoconstriction and impaired oxygenation. Corticosteroid therapy, often used in chronic inflammatory conditions, suppresses fibroblast activity and tensile strength, consistently identified as a risk amplifier in clinical guidelines.[23][21]Advanced age and male gender represent demographic risks for incisional hernia, attributed to age-related tissue fragility and higher rates of abdominal surgeries in men. Patients over 60 years exhibit diminished elastic recoil and slower healing, while male gender confers a hazard ratio of 2.2 for hernia occurrence, possibly due to differences in collagen composition.[20][21]Conditions causing chronic abdominal distension, such as ascites or pregnancy, further exacerbate risk by mechanically stressing the incision site. Ascites, particularly in cirrhotic patients, is associated with a 20% hernia incidence and requires control prior to elective repair. Pregnancy induces similar distension, often rendering asymptomatic hernias symptomatic, with delayed repair recommended unless concurrent with cesarean delivery.[21]
Pathophysiology
Mechanism of Formation
Incisional hernias develop through a multifactorial process involving the failure of multiple layers of the abdominal wall, including the skin, subcutaneous tissue, and particularly the fascia, due to impaired wound healing following surgery. This failure arises from disruptions in the normal healing cascade, where the surgical incision leads to inadequate apposition and reinforcement of the fascial edges, resulting in gaps that are initially filled with weaker scar tissue rather than robust connective tissue. Poor wound healing is exacerbated by local factors such as ischemia or infection, which interrupt the inflammatory, proliferative, and remodeling phases, ultimately compromising the structural integrity of the abdominal wall.[1][24]A key mechanical driver in hernia formation is the increased intra-abdominal pressure that overcomes the weakened fascia, allowing protrusion of peritoneal contents through the defect. This pressure, generated by activities like coughing or straining, acts on the compromised scar tissue, progressively enlarging the fascial gap and forming a hernia sac. The process unfolds in phases: an acute phase immediately postoperative, characterized by wound dehiscence or subtle fascial weakness due to technical or early healing issues; followed by a chronic phase over months to years, where gradual enlargement occurs as ongoing mechanical stress and biological remodeling failures perpetuate the defect.[1][25]At the cellular level, an imbalance in collagen remodeling plays a central role, with reduced deposition of type I collagen in the scar tissue leading to diminished tensile strength. Normally, wound healing involves a shift from type III to type I collagen for durable repair, but in incisional hernias, the type I/III ratio decreases, resulting in immature, less cross-linked extracellular matrix that is prone to deformation. This collagenopathy, combined with dysfunctional fibroblasts and extracellular matrix disorganization, further weakens the repair site.[26][24]Biomechanically, hernia formation typically occurs when the fascial defect exceeds 2-3 cm without adequate reinforcement, as smaller defects may close spontaneously through natural healing, while larger ones persist and widen under physiological loads. This threshold highlights the interplay between defect size and abdominal wall dynamics, where unreinforced gaps beyond this size fail to withstand intra-abdominal forces, leading to irreversible protrusion.[25]
Classification
Incisional hernias are classified according to their anatomical location, defect size, and clinical presentation to facilitate comparison in research, predict complications, and inform surgical strategies.[3]
Location-Based Classification
Incisional hernias are primarily categorized by their location relative to the original surgical incision, which influences recurrence risk and repair techniques. Midline hernias, the most prevalent type, occur along the linea alba following vertical laparotomy incisions and are especially common in the upper abdomen after procedures like gastrectomy or aortic surgery. Paramedian hernias develop adjacent to the rectus sheath from oblique or transverse incisions, while lateral hernias arise from flank or subcostal incisions, such as those used in nephrectomy or cholecystectomy. Specific subtypes include Spigelian incisional hernias, which form near the semilunar line from trocar sites or lateral incisions and are rare but prone to incarceration due to their deep fascial location, and lumbar incisional hernias, which emerge in the posterior abdominal wall after flank surgeries and represent a rare subtype, accounting for less than 1.5% of all abdominal wall hernias, often challenging to diagnose without imaging.[1][27][28][29]
Size-Based Classification
Size classification focuses on the width of the fascial defect, as larger defects correlate with higher complexity and recurrence rates. Incisional hernias are often categorized by size, with small defects typically under 2 cm, frequently occult and detected incidentally; medium defects between 2 and 10 cm, which may present as palpable bulges; and large defects over 10 cm, deemed complex or giant and often requiring advanced reconstructive approaches due to tissue loss and potential loss of domain, though exact criteria vary across sources.[1][30]
Clinical Classification
Clinically, incisional hernias are distinguished as reducible, where protruding contents can be manually returned to the abdominal cavity, or irreducible, signifying incarceration with adhesions that prevent reduction and increase strangulation risk. They are also categorized as symptomatic, manifesting with pain, discomfort, or functional limitation, versus asymptomatic, which may be managed conservatively if small but carry a notable annual risk of complications.[1]
European Hernia Society (EHS) Classification System
The EHS classification, established in 2009, provides a standardized alphanumeric system combining location and width to enhance surgical planning and outcome reporting. Midline locations are subdivided into five zones: M1 (subxiphoidal, from xiphoid to 3 cm caudal), M2 (epigastric, 3 cm below xiphoid to 3 cm above umbilicus), M3 (umbilical, 3 cm above to 3 cm below umbilicus), M4 (infraumbilical, 3 cm below umbilicus to 3 cm above pubis), and M5 (suprapubic, pubic bone to 3 cm cranial). Lateral locations include L1 (subcostal, costal margin to 3 cm above umbilicus), L2 (flank, 3 cm above to 3 cm below umbilicus), and L3 (iliac, 3 cm below umbilicus to inguinal region). Width is graded as W1 (<4 cm), W2 (≥4 cm to <10 cm), or W3 (≥10 cm), allowing descriptors like "M3 W2" for an umbilical defect 4-10 cm wide. This system supports evidence-based guidelines by enabling precise hernia characterization for technique selection, such as mesh reinforcement for W3 defects.[3]
Clinical Features
Signs and Symptoms
Incisional hernias most commonly present with a visible or palpable bulge at the site of a prior abdominal incision, which becomes more prominent during activities that increase intra-abdominal pressure, such as coughing, straining, or performing the Valsalva maneuver.[1][31][32] This bulge may vary in size and is often reducible when the patient is lying down, though it can cause cosmetic concerns as it enlarges over time. Presentation may vary by hernia location (e.g., more noticeable in midline incisions) and patient factors such as obesity, which can obscure the bulge.[1]Patients frequently report localized pain or discomfort at the hernia site, particularly during physical exertion, lifting, or prolonged standing. Small hernias (typically less than 2 cm in diameter) are often asymptomatic or cause only minimal discomfort, while larger hernias exceeding 10 cm more commonly lead to a dull or sharp ache that intensifies with activities like sneezing or bowel movements, potentially resulting in functional impairment such as reduced mobility or avoidance of daily tasks.[31][1] In some cases, overlying skin changes, including thinning, discoloration, or the development of a seroma (a fluid collection under the skin), may occur over the bulge, contributing to further discomfort.[31]A significant proportion of incisional hernias may be asymptomatic or go unnoticed by patients and are discovered incidentally during routine follow-up or unrelated imaging, especially if the defect is small and does not protrude significantly.[33] These cases highlight the progressive nature of the condition, where an initially subtle hernia may worsen gradually, leading to the emergence of symptoms and potential interference with quality of life.[1]
Complications
Incisional hernias can lead to several adverse outcomes if left untreated, ranging from acute emergencies to chronic debilitating conditions. These complications arise primarily from the mechanical effects of abdominal contents protruding through the fascial defect, potentially compromising vascular supply, causing obstruction, or leading to tissue breakdown.[1]Incarceration occurs when hernia contents become trapped and irreducible, often causing severe pain and dysfunction; this affects 6-15% of cases, with higher rates in smaller defects due to tighter entrapment. Adhesions may form around incarcerated contents, further complicating reduction.[34][1]Strangulation represents a life-threatening emergency, where vascular compromise leads to ischemia, necrosis, or perforation of the entrapped tissue; the risk is approximately 2%, necessitating immediate surgical intervention to prevent bowel gangrene or sepsis.[34]Bowel obstruction can result from adhesions or direct pressure by the hernia sac, potentially progressing to perforation if unrelieved; this is a common acute sequela of incarceration, particularly involving small bowel loops.[1]Chronic complications include persistent pain from nerve entrapment or tissue stretching, skin ulceration over large protruding sacs due to chronic pressure and friction, and recurrent infections in the overlying skin or sac from moisture and breakdown.[1]Over time, many incisional hernias enlarge progressively, heightening the risk of incarceration and surgical complexity.[35]
Diagnosis
History and Physical Examination
The history taking for suspected incisional hernia focuses on identifying key elements that support the diagnosis and assess risk. Clinicians inquire about prior abdominal surgeries, as incisional hernias invariably develop at the site of previous incisions.[1] The onset of the bulge is typically reported by the patient, often noticed gradually and becoming more prominent with activities that elevate intra-abdominal pressure, such as straining, coughing, or lifting.[36] Pain patterns are explored, ranging from asymptomatic cases to localized discomfort that worsens with exertion or, in complicated scenarios, severe pain suggesting incarceration.[1] Risk factors like postoperative weight gain, obesity, smoking, malnutrition, and conditions such as diabetes or chronic obstructive pulmonary disease are elicited, as these impair wound healing and increase hernia likelihood.[36][1]Physical examination maneuvers are essential for confirming the presence of an incisional hernia through non-invasive bedside techniques. Inspection and palpation are performed first in the supine position, where the patient is relaxed to identify any subtle bulge or defect at the incision scar; the examiner gently palpates the abdominal wall to delineate the hernia's borders and contents.[36] The patient is then positioned standing to enhance visibility and palpability, as gravity causes the hernia to protrude more noticeably.[1] The cough impulse test follows, with the patient coughing or performing a Valsalva maneuver while the examiner palpates the site; a palpable expansile impulse indicates a fascial defect and supports the hernia diagnosis.[36]Assessment of reducibility is a critical step to evaluate potential complications. The examiner attempts gentle manual reduction of the hernia contents back into the peritoneal cavity; successful reduction suggests a simple hernia, while failure may indicate incarceration, warranting urgent attention.[1] Key examination findings include a soft, compressible tissue mass at the incision site with a positive cough impulse; the overlying skin is inspected for integrity, discoloration, or signs of inflammation, and tenderness is assessed by light pressure, which is typically absent in uncomplicated cases but present if bowel involvement occurs.[36]The sensitivity of physical examination for detecting incisional hernias is approximately 77% overall when compared to computed tomography as the gold standard, with higher reliability in non-obese patients and reduced accuracy (around 73%) in obese individuals or those with small defects, where up to 31% of hernias may be missed.[37] If the examination is inconclusive, particularly in challenging cases, referral for imaging is advised to confirm the diagnosis.[1]
Imaging and Other Tests
Imaging plays a crucial role in confirming the diagnosis of incisional hernia when physical examination is inconclusive, characterizing defect size and contents, assessing for complications such as incarceration, and aiding preoperative planning.[1][9]Ultrasound serves as a first-line imaging modality for suspected superficial incisional hernias, offering dynamic evaluation during patient straining or Valsalva maneuver to detect defects and protruding contents in real time. It exhibits sensitivity ranging from 71% to 98% and specificity from 88% to 100% when compared to CT as the reference standard.[9][38]Computed tomography (CT) is the gold standard for diagnosing incisional hernias, particularly in complex cases involving obesity or suspected complications, as it precisely delineates defect dimensions, hernia sac contents, multifocal involvement, and surrounding anatomy while minimizing radiation exposure through targeted protocols. CT demonstrates a sensitivity of 100% and specificity of 98% relative to intraoperative confirmation.[1][9][38]Magnetic resonance imaging (MRI) is infrequently utilized due to its higher cost and scan duration but proves valuable for detailed soft tissue assessment in obese patients or when ionizing radiation from CT is contraindicated.[1][9]Additional tests such as herniography—involving peritoneal contrast injection—are rarely employed for incisional hernias owing to their invasiveness and limited availability, while diagnostic laparoscopy may identify occult defects not apparent on noninvasive imaging.[39][38]These imaging techniques facilitate differentiation of incisional hernias from mimics like hematomas or tumors and reveal multifocal or incarcerated defects overlooked during clinical assessment alone.[39][38]
Treatment
Conservative Management
Conservative management of incisional hernia focuses on non-surgical strategies for monitoring and symptom alleviation, particularly in cases where surgical intervention poses excessive risk or the hernia presents minimal threat. Watchful waiting is appropriate for small, asymptomatic hernias, with patients undergoing annual clinical evaluations to assess for changes in size, symptoms, or complications such as incarceration. This approach carries a low annual risk of acute events, estimated at approximately 2.6%, allowing for safe observation in select patients.[1]Supportive measures play a key role in symptom control and risk mitigation during conservative management. Weight loss is strongly recommended for obese patients, as excess body mass exacerbates intra-abdominal pressure and hernia progression; activity modification, including avoidance of heavy lifting, further reduces strain on the abdominal wall. For discomfort, non-steroidal anti-inflammatory drugs (NSAIDs) or simple analgesics provide effective pain relief in the absence of obstruction or strangulation. These interventions are particularly indicated for patients with high surgical comorbidities or small, low-risk defects where the benefits of observation outweigh operative risks.[1][40][41]Despite these benefits, conservative management has notable limitations and is unsuitable for symptomatic, enlarging, or complicated hernias. Natural progression occurs in a significant proportion of cases, with up to 22% of patients transitioning to surgical need within the first few months of watchful waiting and an additional 10% over the subsequent years, underscoring the importance of vigilant monitoring to detect enlargement or acute deterioration early. This strategy is thus reserved for stable, reducible hernias in medically optimized individuals, with prompt escalation to surgery if symptoms worsen or quality of life is impaired.[42]
Surgical Repair
Surgical repair serves as the definitive treatment for incisional hernias, particularly in cases where conservative measures have failed or acute complications arise. Open techniques are widely employed for their direct access to the defect, while minimally invasive approaches have gained prominence for suitable candidates. Mesh reinforcement is standard in most repairs to achieve tension-free closure and minimize recurrence. Recent European Hernia Society (EHS) guideline protocols as of 2025 emphasize evidence-based approaches, including emerging preoperative optimizations like botulinum toxin A injections for large defects to facilitate closure.[25][43]In open repair, the sublay technique—also termed retrorectus or Rives-Stoppa repair—positions synthetic mesh in the preperitoneal space posterior to the rectus abdominis muscle, ensuring at least 5 cm overlap beyond the defect edges. This method isolates the mesh from intra-abdominal contents, promoting tissue ingrowth and reducing recurrence to less than 10% with proper placement.[25][44] Alternatively, onlay mesh placement involves suturing the mesh over the anterior fascia following primary defect closure, though it is associated with higher rates of superficial wound complications compared to sublay due to its subcutaneous position.[44][42]Laparoscopic and robotic-assisted repairs utilize the intraperitoneal onlay mesh (IPOM) technique, where a large prosthesis is deployed and secured intraperitoneally using transabdominal sutures and tacks, with 4–5 cm overlap to distribute intra-abdominal forces evenly. These minimally invasive methods shorten hospital stays (typically 2–3 days versus 4–5 days for open repair) and accelerate recovery by limiting tissue trauma and postoperative pain.[45][46] The IPOM-plus variant incorporates primary fascial closure prior to mesh fixation to further mitigate seroma formation and recurrence.[25]Mesh selection is guided by defect size, infection risk, and surgical field contamination. Synthetic nonabsorbable meshes, such as macroporous polypropylene (weight 35–90 g/m²), are the first-line choice for clean or clean-contaminated cases due to their strength, biocompatibility, and cost-effectiveness in achieving durable reinforcement for defects of varying sizes.[25] Biologic meshes, derived from decellularized xenogeneic or allogeneic tissues, are preferred in contaminated or infected fields to reduce the risk of mesh-related infections, though they may be more suitable for smaller defects or bridging scenarios.[25] Absorbable synthetic meshes, including those based on polyglycolic acid or poly-4-hydroxybutyrate, provide temporary scaffold support and are considered for high-risk patients or when long-term reinforcement is unnecessary, pending further outcome data.[25][47]For complex hernias involving large defects exceeding 10 cm or loss of domain, component separation techniques enable tension-free primary closure by releasing muscular layers. The anterior approach mobilizes the external oblique for 4–8 cm advancement, while the posterior transversus abdominis release (TAR) accesses the retrorectus space bilaterally for similar gain, often combined with mesh reinforcement.[25] In cases where fascial approximation is infeasible, bridging with mesh spans the defect, though this carries a higher tension profile.[25]Emergency surgical repair is mandated for strangulated incisional hernias to prevent bowel ischemia, with immediate exploration via open or laparoscopic access depending on stability. If necrotic bowel is identified, resection and anastomosis precede hernia correction, using biologic or no mesh in contaminated settings to avoid exacerbating infection.[48][49][42]
Prognosis and Prevention
Outcomes and Recurrence
The prognosis for incisional hernia repair is generally favorable when performed electively on symptomatic or large defects, with mesh reinforcement significantly reducing recurrence compared to suture-only techniques. Recurrence rates after mesh-augmented repair typically range from 10% to 20% at 5 years, though they can reach 18-20% at 15 years in nationwide cohorts, and up to 30% in high-risk cases involving multiple prior repairs. Laparoscopic approaches may yield slightly lower cumulative recurrence risks (around 15%) compared to open repairs (21%) over medium-term follow-up.[50][51][52]Key factors influencing recurrence include patient-related risks such as obesity (body mass index >30 kg/m²), smoking, and large defect sizes (>20 cm² estimated area), as well as surgical elements like inadequate mesh overlap (<7 cm) or bridging without closure. Technical failures, such as tension on the repair or postoperative infections, further elevate 5-year recurrence to 23-35% in reoperative cases. In high-risk groups, like those with contaminated fields or multiple recurrences, rates can exceed 24% even with advanced techniques.[50][53][54]Surgical repair markedly improves quality of life in most patients, with 80-90% reporting reduced pain, better abdominal function, and alleviation of symptoms like bulging or discomfort within 12 months postoperatively. Studies using validated tools like the EQ-5D index show mean improvements of up to 89% from preoperative baselines, shifting from poor/very poor quality of life in nearly half of cases to good/very good in over 80%. However, persistent issues like chronic pain can affect 10-20% of patients long-term.[55][56]Postoperative morbidity remains a concern, with surgical site infections occurring in 5-10% of open mesh repairs (lower at 1-3% for laparoscopic), seromas in 8-12% overall (up to 30-50% in some mesh placements), and mesherosion or migration as rarer events (1-3%) linked to chronic complications like fistulas. These events can prolong recovery but are manageable in most cases without impacting overall prognosis. Follow-up typically involves clinical examination and optional imaging (e.g., ultrasound or CT) at 1-2 years to detect early recurrence, with longer-term monitoring recommended given rising risks beyond 5 years.[57][58][59]
Preventive Strategies
Preventing incisional hernias involves evidence-based techniques focused on surgical closure, patient optimization, and postoperative management to minimize abdominal wall stress and promote healing. Optimal fascial closure during laparotomy is critical, with recommendations emphasizing the use of long-acting absorbable monofilament sutures, such as polydioxanone, to provide sustained tensile strength. [1] A key technical aspect is maintaining a suture-to-wound length ratio of at least 4:1 using the "small bites" technique, where bites are 5-8 mm apart and 5-8 mm from the wound edge, which has been shown to significantly reduce incisional hernia rates compared to traditional large bites. [1][60]In high-risk patients, such as those undergoing emergency laparotomy, prophylactic mesh augmentation during closure is recommended to distribute tension and reinforce the abdominal wall. Techniques like retrorectus or onlay mesh placement have demonstrated substantial reductions in incisional hernia incidence, with meta-analyses reporting rates as low as 11-13% with mesh versus 21-40% without, representing approximately a 50% relative risk reduction in high-risk scenarios. [61][62] Permanent synthetic meshes are preferred in clean cases, while absorbable or biologic options may be used in contaminated fields to mitigate infection risks. [1]Perioperative care plays a vital role in addressing modifiable risk factors. Strict infection control measures, including prophylactic antibiotics and aseptic techniques, are essential to prevent surgical site infections that can weaken the fascia and lead to hernia formation. [63] Preoperative smoking cessation for at least 4 weeks has been associated with reduced wound complications and lower incisional hernia risk by improving tissue oxygenation and collagen synthesis. [57] Similarly, nutritional optimization, such as ensuring adequate protein intake and correcting malnutrition, supports wound healing and is particularly important in patients with low albumin levels. [64]Surgical approach selection can further lower incidence rates. Minimally invasive techniques, like laparoscopy, reduce tissue trauma and are linked to fewer incisional hernias compared to open procedures. [65] When open surgery is required, transverse incisions, such as Pfannenstiel or periumbilical transverse, are preferable to midline laparotomy due to better alignment with muscle fibers and lower hernia rates, with studies showing up to a 50% reduction in risk. [66][67]Postoperatively, early mobilization within 24-48 hours encourages circulation and prevents deconditioning without increasing hernia risk, as part of enhanced recovery protocols. [68] Wound support measures, including abdominal binders or negative pressure wound therapy, provide external reinforcement to minimize intra-abdominal pressure during coughing or straining, thereby aiding fascial approximation and reducing early dehiscence. [69][70]