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Post hoc

Post hoc, formally known as —a Latin phrase translating to "after this, therefore because of this"—is a that occurs when a assumes a causal between two events solely because one precedes the other in time. The term is also used in to refer to analyses or tests conducted after initial data examination or testing. This error confuses temporal sequence with causation, ignoring other potential factors or explanations that could link the events. As a subtype of false cause fallacies, it highlights the dangers of hasty generalizations in reasoning, where is mistaken for direct influence without supporting evidence. Later thinkers, such as in his System of Logic, critiqued it as a form of overgeneralization that overlooks multiple contributing causes. In modern , it is classified among causal paralogisms, often appearing in arguments that fail to demonstrate a mechanistic process beyond mere chronology. For instance, claiming that a policy change caused an economic shift simply because the shift followed the change exemplifies this error, as alternative variables like market trends may be at play. Recognizing the fallacy is essential in , scientific inquiry, and everyday discourse to avoid misleading conclusions. It frequently surfaces in , political , and , where proponents attribute outcomes to prior actions without rigorous testing. To counter it, arguments must provide empirical data establishing causation, such as controlled experiments or statistical controls for variables. By distinguishing from cause, individuals can foster more accurate and reliable interpretations of events.

Etymology and General Meaning

Literal Translation

The Latin phrase post hoc directly translates to "after this," where post signifies "after" or "subsequent to," and hoc is the neuter accusative singular form of the pronoun hic, haec, hoc, denoting "this" or "the thing." This construction in Latin typically indicates temporal or sequential relation, referring to an event or following a specified one. In English adoption, post hoc retains this core meaning of "after this" or "subsequent," often implying retrospection or occurrence following a prior . It entered English by the early , with the first attested use in , used standalone to describe matters arising or addressed after the fact. The phrase is pronounced /poʊst ˈhɑːk/ in standard English contexts, approximating with a on the second word.

Historical Usage

The phrase "post hoc," meaning "after this" in Latin, first appears in classical Latin literature as a standard expression denoting temporal sequence. It is attested in the works of Marcus Tullius Cicero, the Roman statesman and philosopher active in the 1st century BCE, where it is employed in rhetorical and philosophical contexts to describe events following one another without implying causal relationships. The adoption of "post hoc" into English occurred gradually through scholarly and legal channels, reflecting the influence of Latin on early modern intellectual traditions. This usage emphasized sequence in judicial reasoning without presuming causation, aiding in the clarification of timelines in litigation. In non-fallacious contexts, "post hoc" appeared in 18th-century philosophy texts to describe mere chronological succession in historical or empirical narratives. This neutral application persisted in philosophical writing to avoid erroneous inferences, focusing instead on descriptive chronology. By the , interpretations began shifting toward recognizing potential fallacious implications in certain contexts.

Logical Fallacy

Definition of Post Hoc Ergo Propter Hoc

Post hoc ergo propter hoc is a Latin phrase that literally translates to "after this, therefore because of this." This expression denotes an informal logical in which the temporal sequence of two events is mistakenly interpreted as of a causal relationship between them. At its core, the fallacy occurs when one assumes that because event A precedes event B in time, event A must have caused event B, without additional demonstrating a causal or accounting for potential confounding factors. This error represents a failure in , where chronological order is conflated with causation. Classified as a subtype of fallacies, belongs to the broader category of informal fallacies, which involve defects in reasoning content or context rather than violations of formal deductive validity. Unlike formal fallacies, such as , it arises in arguments and inductive inferences, highlighting a disconnect between premises and conclusion without structural invalidity in syllogistic form. The essential logical structure of the fallacy can be outlined as follows: event A occurs, followed by event B; therefore, A caused B. This inference lacks justification unless supported by empirical data on mechanisms, correlations, or experimental controls.

Mechanisms and Common Errors

The post hoc ergo propter hoc fallacy stems from cognitive biases that predispose individuals to infer causation from temporal sequence alone. Confirmation bias plays a key role, as people selectively attend to and recall evidence that aligns with an assumed causal relationship while disregarding disconfirming instances, thereby reinforcing erroneous beliefs about event sequences. Illusory correlation further contributes by leading individuals to overestimate associations between unrelated events, particularly when those events co-occur frequently or saliently, fostering a perception of patterned causality in random or coincidental sequences. These biases are rooted in the brain's tendency to seek explanatory patterns for uncertainty, a heuristic that aids survival but falters in complex causal reasoning. Common errors in committing the fallacy include overlooking alternative causes, where intervening or factors are ignored in favor of the temporally prior event; reverse causation, in which the assumed effect is mistakenly treated as the cause; and coincidental timing, attributing significance to chance alignments without verifying independence. Such mistakes often arise from failing to rigorously evaluate causal claims against established frameworks, such as the , which stress that mere temporal precedence is insufficient for inferring causation without supporting evidence like biological plausibility and dose-response relationships. In rhetorical contexts, the is frequently deployed in arguments, speeches, and media narratives to imply through sequential event descriptions, exploiting audience tendencies toward to persuade without empirical validation. This simplifies complex issues by chaining events narratively, as seen in persuasive where prior occurrences are framed as direct precursors to outcomes, bypassing the need for mechanistic explanation. Detecting the post hoc fallacy involves applying a structured for causal validity: confirm temporal order as a , but supplement it with evidence of consistent , a plausible underlying , and replicability across independent observations to rule out spurious links. The offer a comprehensive guide, evaluating aspects like strength, specificity, and coherence to differentiate genuine causation from temporal artifacts, thereby mitigating errors in inference.

Historical Examples

In , superstitions often led to the , where preceding omens were blamed for subsequent calamities like plagues, as people attributed natural disasters to divine wrath without evidence of causation. The poet critiqued this in his epic (c. 55 BCE), arguing that fear of gods and omens oppressed humanity, using to explain phenomena naturally rather than through supernatural sequences. For instance, in Book VI, he described the Athenian plague of 430 BCE, detailing failed religious rituals and attributing the outbreak to environmental factors like contaminated winds, rejecting the idea that prior portents caused it as mere . During the medieval period, celestial events were frequently interpreted as causal precursors to political upheavals, exemplifying the fallacy in historical chronicles. The appearance of in 1066 was seen as an ill omen preceding the , with contemporary accounts like the depicting it as a harbinger of King Harold II's defeat at the on October 14, 1066, implying the comet directly influenced the outcome despite no causal link. English and Norman sources, including the , reinforced this by linking the comet's visibility in April-May 1066 to the ensuing royal deaths and invasion, a view perpetuated in medieval where temporal sequence was mistaken for causation. In the , philosophers began systematically identifying the within , highlighting its pitfalls in scientific and everyday . addressed it explicitly in (1843), classifying "" as a common error where mere temporal succession is assumed to prove causation, using examples from historical events and experiments to warn against uncritical generalizations. Mill emphasized that while sequence may suggest , it requires additional like joint variation or elimination of alternatives to establish true cause, critiquing its role in flawed historical narratives and policy decisions. Early 20th-century political frequently exploited the to attribute economic downturns to prior policies or events, fostering without substantiation. In Weimar Germany, Nazi rhetoric post-1919 blamed the for the of 1923 and the Great Depression's onset in 1929, portraying the treaty's as the direct cause of and poverty despite intervening factors like global trade collapse. This narrative, disseminated through speeches and publications like ' writings, assumed sequence implied causation to rally support, ignoring evidence that domestic fiscal policies and international lending played larger roles.

Statistical Applications

Post Hoc Analysis in Hypothesis Testing

In statistical hypothesis testing, refers to exploratory procedures conducted after an initial , such as analysis of variance (ANOVA), reveals a significant overall effect, allowing researchers to investigate specific patterns or differences in the observed data. These analyses are typically unplanned and retrospective, focusing on data that has already been collected and examined, rather than being specified in advance based on prior theory. The primary purpose of is to pinpoint which particular groups or variables contribute to the detected significance, thereby facilitating the generation of new hypotheses for future research without assuming definitive causation from the exploratory findings alone. For instance, after an ANOVA indicates differences among treatment groups, post hoc methods can reveal pairwise comparisons that were not hypothesized a priori, aiding in the refinement of scientific understanding. This approach is valuable for uncovering unexpected relationships but requires cautious interpretation to avoid overgeneralization. Post hoc analysis is commonly applied in experimental designs, including randomized controlled trials (RCTs) where initial results prompt deeper subgroup explorations, and in observational studies to identify emergent patterns in large datasets. In RCTs, for example, it might examine differential effects across unexpected subgroups, such as varying doses or demographics, following primary outcome assessments. Unlike the logical fallacy of , which erroneously infers causation solely from temporal sequence, statistical employs controlled, validated techniques to explore data retrospectively while emphasizing the need for subsequent confirmatory studies to establish reliability. This distinction underscores its role as a hypothesis-generating tool rather than a source of unsubstantiated causal claims.

Specific Post Hoc Tests

After an ANOVA indicates significant differences among group means, specific post hoc tests are employed to identify which pairs or contrasts differ. Tukey's Honestly Significant Difference (HSD) test is a widely used procedure for conducting all pairwise comparisons between group means when sample sizes are equal, controlling the at the desired level. The test computes the difference between each pair of means and compares it to a given by q_{\alpha, k, \nu} \sqrt{\frac{MSE}{n}}, where q_{\alpha, k, \nu} is the studentized range for significance level \alpha, k is the number of groups, \nu is the for the error term, MSE is the error from the ANOVA, and n is the common sample size per group; if the absolute difference exceeds this value, the means are declared significantly different. Scheffé's method provides a more conservative approach suitable for examining all possible linear among group means, rather than just pairwise comparisons, and is particularly useful when the researcher has no predefined contrasts in mind. It adjusts the F-statistic for , where the for a contrast estimate is based on \sqrt{(k-1) F_{\alpha, k-1, \nu}} times the of the contrast, with F_{\alpha, k-1, \nu} being the critical F-value from the with k-1 and \nu ; this ensures control over the for any set of contrasts. The method's conservatism arises from protecting against all possible contrasts, making it less powerful for specific pairwise tests but more versatile for exploratory analyses. The Bonferroni correction offers a straightforward adjustment for multiple comparisons by dividing the overall significance level \alpha by the number of tests m, yielding an adjusted level \alpha' = \alpha / m, which is then applied to each individual test (such as t-tests) to maintain the family-wise error rate. This simple inequality-based procedure, while conservative especially for large m, is easy to implement and applicable to any set of hypothesis tests, including post hoc comparisons after ANOVA. These post hoc tests share common assumptions derived from ANOVA, including normality of the residuals within each group and homogeneity of variances across groups, which can be checked using diagnostic plots or . Tukey's HSD is preferred when sample sizes are equal and the focus is on all pairwise comparisons due to its balance of power and error control, while Scheffé's method is chosen for complex contrasts or unequal sample sizes despite its lower power, and the is ideal for a small, planned set of comparisons or when computational simplicity is prioritized.

Addressing Multiple Comparisons

The in post hoc testing occurs when multiple hypothesis tests are performed on the same dataset, substantially increasing the (FWER)—the probability of incurring at least one Type I error (false positive) across the entire set of tests. For example, conducting 10 independent tests each at a level of α = 0.05 yields a probability of approximately 0.40 for at least one false positive, as calculated by 1 - (1 - α)m, where m is the number of tests./06%3A_Multiple_Tests/6.01%3A_Multiple_Comparisons) This inflation arises because the nominal α level applies per test, but the cumulative risk escalates with more comparisons, undermining the reliability of exploratory findings./06%3A_Multiple_Tests/6.01%3A_Multiple_Comparisons) To address this, methods beyond basic corrections like Bonferroni or Tukey are employed, with the (FDR) providing a more powerful framework by targeting the expected proportion of false positives among significant results rather than strictly bounding all errors. The seminal Benjamini-Hochberg procedure controls FDR by sorting the m p-values in ascending order p_{(1)} \leq p_{(2)} \leq \cdots \leq p_{(m)} and rejecting all null hypotheses up to the largest i where p_{(i)} \leq \frac{i}{m} q with q denoting the target FDR level; this monotonic approach ensures FDR ≤ q under independence or positive dependence of test statistics. Effective management of post hoc analyses also involves best practices such as defining pre-planned contrasts a priori, grounded in theoretical predictions, to limit the scope of comparisons and preserve statistical power without necessitating broad corrections. Complementing this, reporting standardized effect sizes—like Cohen's d, which quantifies the mean difference in standard deviation units—offers insight into practical significance and helps interpret the magnitude of detected effects. Furthermore, replicating post hoc findings in independent datasets is crucial for establishing their robustness, as initial exploratory results are prone to capitalization on chance. Criticisms of multiple comparison corrections highlight their potential over-conservatism, particularly with stringent FWER methods, which can reduce and increase Type II s by failing to detect true effects in sparse-signal scenarios. Balancing this requires integrating to evaluate trade-offs, ensuring study designs maintain sufficient to meaningful differences while controlling rates.

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