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PBB

Polybrominated biphenyls (PBBs) are a class of synthetic brominated hydrocarbons featuring a central core substituted with one to ten atoms, manufactured primarily as additive retardants for plastics, textiles, synthetic fibers, and to enhance resistance. Their high stability, , and resistance to degradation enable in fatty tissues and in soils and sediments for decades. PBB production and use peaked in the mid-20th century but ceased in the United States following voluntary phase-out and regulatory bans due to concerns, with hexabromobiphenyl (FireMaster) being the dominant commercial formulation. The compounds entered the global spotlight through the 1973 contamination incident, where a labeling error at a Michigan Chemical Corporation facility led to 500 pounds of FireMaster being shipped instead of , resulting in its inadvertent mixing into cattle feed supplements distributed statewide. This error contaminated over 100 farms' livestock, dairy products, eggs, and meat, exposing an estimated 90% of 's population via the before detection in 1974, prompting mass quarantines, the slaughter of thousands of animals, and farm buyouts costing millions. Empirical studies of the cohort, tracking over 3,800 exposed individuals since the , have documented elevated PBB levels in blood and correlating with acute symptoms like , , and appetite loss, alongside potential long-term risks including altered immune function, disruption, and reproductive effects, though definitive causal links to cancer or widespread morbidity remain inconclusive amid variables and ethical limitations. The episode highlighted failures in industrial , regulatory oversight, and , fueling debates on precautionary principles versus empirical in , with PBBs now serving as a in persistent organic pollutant management under frameworks like the Convention.

Chemical Structure and Properties

Molecular Composition and Isomers

Polybrominated biphenyls (PBBs) consist of a core structure, comprising two linked phenyl rings (C12H10), in which 1 to 10 atoms replace atoms, yielding the general molecular formula C12H(10-n)Brn where n ranges from 1 to 10. These compounds are synthetic organobromines, with substitution occurring at various positions on the aromatic rings, influencing their physical properties and biological behavior. The structural variability results in 209 possible congeners, each defined by the specific number and positional arrangement of atoms across the skeleton. Congeners are systematically numbered from 1 to 209 following conventions similar to those for polychlorinated biphenyls, prioritizing the lowest sets for substitution patterns. For instance, the hexa-substituted congener BB-153 corresponds to 2,2',4,4',5,5'-hexabromobiphenyl (C12H4Br6), while BB-209 denotes decabromobiphenyl (C12Br10). The number of isomers increases with count: monobromobiphenyl has 3, dibromobiphenyl 12, up to hexabromobiphenyl with 42, after which it decreases symmetrically to 1 for decabromobiphenyl. Commercial PBB formulations were typically mixtures dominated by higher-brominated congeners, such as hexabromobiphenyls (e.g., FireMaster BP-6 containing primarily BB-153 and related hexa-isomers), octabromobiphenyls, and decabromobiphenyl (BB-209). These mixtures reflected processes favoring symmetric or near-symmetric substitutions for stability and efficacy as flame retardants, though trace lower-brominated congeners could form impurities. Congeners lacking ortho-bromine substituents (coplanar forms) exhibit dioxin-like planarity, potentially enhancing , whereas ortho-substituted variants are more twisted and less persistent in some biological contexts. Analytical often relies on gas chromatography-mass spectrometry to distinguish isomers based on retention times and fragmentation patterns unique to distributions.

Physical and Chemical Characteristics

Polybrominated (PBBs) consist of a core substituted with 2 to 10 atoms, resulting in 209 possible congeners, though commercial mixtures primarily feature hexa- and octa-brominated forms. These compounds appear as white to off-white crystalline solids or powders at ambient temperatures. Physical properties vary by congener and bromination degree; for instance, hexabromobiphenyl isomers exhibit melting points ranging from 72 °C to approximately 223–250 °C, while commercial products like FireMaster BP-6 (predominantly hexabromobiphenyl) have reported melting ranges of 200–250 °C. points are high, often exceeding 450 °C, with many congeners decomposing before reaching them. PBBs demonstrate low volatility, with vapor pressures typically between 5.2 × 10^{-8} mm and 7 × 10^{-11} mm at 25 °C, decreasing as bromine substitution increases. PBBs are highly hydrophobic and lipophilic, with water solubilities generally below 0.011 mg/L at 25 °C, rendering them insoluble in aqueous media but highly soluble in nonpolar organic solvents such as , , , and acetone. Octanol-water partition coefficients (log K_{ow}) range from 5.53 to 9.10 across congeners, underscoring their affinity for and potential for . Chemically, PBBs exhibit high thermal and oxidative , resisting ignition and under standard conditions, which facilitated their use as retardants. They are inert to and most but susceptible to slow under light or anaerobic microbial debromination.
PropertyTypical Range/ValueExample Congener/Reference
Melting Point72–250 °CHexabromobiphenyl: 72–223 °C; FireMaster BP-6: 200–250 °C
10^{-8} to 10^{-11} mm (25 °C)Hexabromobiphenyl: 5.2 × 10^{-8} mm
Solubility<0.011 mg/L (25 °C)General for PBBs
Log K_{ow}5.5–9.1Varies by congener; e.g., 6.39 for common hexabromo

Production and Commercial Use

Manufacturing Processes

Polybrominated biphenyls (PBBs) are manufactured through the electrophilic aromatic substitution reaction known as bromination of biphenyl, where biphenyl reacts with bromine to introduce multiple bromine atoms onto the aromatic rings. This process yields mixtures of congeners rather than single compounds, with the degree of bromination controlled by reaction parameters such as bromine excess, temperature, and catalyst choice. The reaction typically employs Friedel-Crafts-type conditions, using Lewis acid catalysts like aluminum chloride (AlCl₃), aluminum bromide (AlBr₃), or iron to generate the electrophilic brominating species. is often dissolved in an organic solvent such as ethylene dibromide, and bromine is added in 0–20% excess to promote higher bromination levels, resulting in predominantly hexa-, octa-, nona-, or deca-brominated products depending on the target mixture. For specifically, excess bromine serves dual roles as reactant and solvent, with the catalyst facilitating complete substitution; post-reaction, unreacted bromine is distilled using a brominated solvent for purification. Commercial production, which occurred primarily in the United States from 1970 to 1979, utilized batch processes in corrosion-resistant reactors equipped with acid scrubbers to handle hydrogen bromide byproducts. Water was added to the reaction mixture to quench the process, generating effluent discharged to surface waters, while gaseous emissions were captured via vents. Losses during manufacturing included approximately 1,125 pounds of PBBs to air and 0.0046 pounds to water per million pounds produced. Laboratory-scale alternatives, such as diazo coupling of brominated anilines with bromobenzenes, were not used commercially. Production ceased in the U.S. following the and regulatory scrutiny.

Historical Applications as Flame Retardants

Polybrominated biphenyls (PBBs) were introduced as commercial additive flame retardants in the United States around 1970, primarily to enhance fire resistance in various polymeric materials by suppressing or delaying combustion. The primary product, , consisted mainly of hexabromobiphenyl congeners (over 50% 2,2',4,4',5,5'-hexabromobiphenyl), produced by in St. Louis, Michigan. This formulation was incorporated at concentrations typically ranging from 10-15% by weight into acrylonitrile-butadiene-styrene (ABS) thermoplastics for applications such as housings for televisions, business machines (e.g., typewriters and calculators), and electronic equipment. Total U.S. production of PBBs reached approximately 13.3 million pounds between 1970 and 1976, with hexabromobiphenyl accounting for about 88% (11.8 million pounds). Additional PBB variants, including and , were manufactured by companies such as White Chemical Company and Hexcel Corporation until 1979, often under trade names like Bromkal 80 or Flammex B 10 in European markets. These higher-brominated forms were applied in polyurethane foams for automotive upholstery, synthetic fibers, molded plastics, coatings, and lacquers, providing thermal stability and reduced flammability in industrial and consumer products. , a variant containing 2% calcium silicate, was similarly used but became notorious due to its accidental contamination of livestock feed in 1973. PBBs were valued for their compatibility with additives and effectiveness in end-use products without significantly altering material properties. Commercial use of PBBs declined sharply following the 1973-1974 Michigan contamination incident, which exposed widespread environmental and health risks, leading to voluntary discontinuation by manufacturers. By 1979, domestic production had ceased entirely, with remaining stocks phased out and applications largely replaced by in flame-retardant formulations. Despite their efficacy, PBBs were not chemically bound to host polymers, facilitating potential migration and persistence, though this was not fully appreciated at the time of adoption. No significant imports or exports occurred post-1979 in the U.S., marking the end of their historical role in flame retardation.

Toxicological Profile

Acute and Subchronic Effects

PBBs exhibit low acute toxicity across exposure routes, with oral LD50 values exceeding 10 g/kg body weight in rats for commercial mixtures like FireMaster BP-6. In single high-dose oral studies in rats (up to 1,000 mg/kg), effects were limited to transient body weight reduction and reduced food intake, without histological liver changes or overt mortality at non-lethal levels. Dermal application in rabbits caused mild skin irritation and increased liver weight with at higher doses, but no systemic lethality. Inhalation studies in animals showed no significant adverse effects, though data are limited to two studies with commercial mixtures. Subchronic effects, observed in repeated oral dosing over 14–90 days, primarily target the liver and in . In rats dosed at 1–3 mg/kg/day, liver weights increased with enlargement and induction, while weights rose and T4 levels decreased, indicating disruption of . NOAELs for these hepatic and effects were around 1 mg/kg/day, with LOAELs at 3 mg/kg/day. Immunological alterations, including reduced weight and lymphoproliferative responses, emerged at 1 mg/kg/day in rats. Body weight reductions and feed intake suppression were consistent across studies, often preceding organ-specific toxicities. In mice, similar hepatic effects occurred at 0.3 mg/kg/day, with atrophy at higher subchronic doses. No directly address subchronic PBB , but models suggest sensitivity in at lower doses (e.g., 0.012 mg/kg/day for ).

Mechanisms of Toxicity and Bioaccumulation

Polybrominated biphenyls (PBBs) primarily exert toxicity through activation of the aryl hydrocarbon receptor (AhR), mimicking dioxin-like effects in experimental models. This receptor-mediated pathway induces cytochrome P450 enzymes, such as CYP1A isoforms, which enhance oxidative metabolism and can disrupt endogenous processes like vitamin A homeostasis and porphyrin synthesis. Specific congeners, including hexabromobiphenyl (FireMaster FF-1 component), bind AhR with varying affinity, leading to downstream effects like thymic atrophy, hepatotoxicity, and altered immune function observed in rodent studies at doses as low as 0.3 mg/kg/day. While the precise molecular interactions differ by congener bromination degree, enzyme induction correlates with lateral bromine substitutions, facilitating AhR transformation and DNA binding. Additional toxicity arises from endocrine disruption, particularly interference with hormone regulation, as bromine atoms compete with iodine in enzymes, reducing circulating thyroxine levels in exposed animals by up to 50% at chronic doses of 10 mg/kg. This mechanism contributes to developmental delays and reproductive impairments, with intergenerational effects noted in offspring from dams exposed to 100 mg/kg during . Hepatocellular effects, including and neoplasia promotion, stem from sustained microsomal enzyme proliferation rather than direct , as PBBs show weak mutagenicity in Ames assays. PBBs bioaccumulate due to high ( Kow values exceeding 7 for congeners) and to metabolic degradation, preferentially partitioning into with bioconcentration factors up to 10^4 in aquatic organisms. Oral exceeds 90% in mammals, followed by distribution to lipid-rich organs like liver and , where unchanged parent compounds predominate due to limited or debromination. Elimination is protracted, with half-lives estimated at 10–11 months post-exposure, though adipose retention may extend effective persistence to years, as evidenced by detectable levels in cohort serum decades after the 1973 incident. occurs across trophic levels, with concentrations increasing 5–10-fold from feed to dairy products in contaminated livestock.

Evidence on Carcinogenicity and Long-Term Health Outcomes

Animal studies demonstrate that polybrominated biphenyls (PBBs), particularly mixtures like FireMaster FF-1, induce hepatocellular carcinomas and cholangiocarcinomas in rats and mice of both sexes following oral exposure, with doses ranging from 0.5 mg/kg/day over 115 weeks to single gavage doses of 1,000 mg/kg. Perinatal and chronic dietary exposures further increase liver tumor incidence and multiplicity in these species. PBBs exhibit non-genotoxic mechanisms, acting as tumor promoters via aryl hydrocarbon receptor (AhR) activation, with congener-specific effects such as hepatic promotion by hexabromobiphenyl congeners. The National Toxicology Program (NTP) classifies PBBs as reasonably anticipated to be human carcinogens based on this sufficient evidence from experimental animals, while the International Agency for Research on Cancer (IARC) designates them as probably carcinogenic to humans (Group 2A), citing inadequate human data but strong mechanistic parallels to polychlorinated biphenyls (PCBs). Human epidemiological evidence remains limited and inconclusive, primarily derived from the 1973 contamination exposed via contaminated feed. Case-control analyses within this population reported elevated odds ratios for (OR=3.3, 95% CI=0.9-11.4 at PBB ≥2 ppb) and digestive system cancers (OR=22.9, 95% CI=1.34-392 at >50 ppb) after approximately 20 years latency, though small sample sizes and exposure misclassification limit reliability. A 2025 follow-up of the Long-Term PBB (enrollment PBB medians of 3-4 µg/L) found no with all-cause mortality (female HR=1.03, 95% CI=0.84-1.25 for high exposure; male HR=0.95, 95% CI=0.80-1.13), but noted increased cancer mortality in females (HR=1.50, 95% CI=1.02-2.22), particularly respiratory cancers (HR=3.82, 95% CI=1.38-10.61), with reduced risk in males (HR=0.68, 95% CI=0.46-1.01). No consistent links to other cancers or circulatory mortality emerged, and modified some risks in males. Beyond carcinogenicity, long-term PBB exposure in the cohort and occupational settings correlates with endocrine disruption, including and altered levels (e.g., reduced T4/T3, elevated thyrotropin in 4/35 exposed workers). Animal models confirm follicular hyperplasia and enlargement at doses ≥0.3 mg/kg/day, alongside persistent hepatic effects like enlargement and at ≥0.1 mg/kg/day. Reproductive outcomes show associations with alterations in utero-exposed females, while dermal effects such as bromacne affected 70% of production workers with mean serum PBB of 603.9 ppb. Neurological reports include in exposed residents without clear dose-response, and neurobehavioral changes in offspring at maternal doses ≥0.2 mg/kg/day. PBBs' long (e.g., 6-15 years for hexabromobiphenyl congeners) contributes to sustained body burdens, potentially amplifying chronic risks despite low-level exposures. Overall, while animal data robustly support multi-organ toxicity, human findings are confounded by co-exposures and require further longitudinal validation.

Environmental Fate and Exposure Pathways

Persistence and Degradation

Polybrominated biphenyls (PBBs) exhibit high environmental persistence owing to their chemical stability, resistance to , and limited susceptibility to biotic and abiotic processes. In soils, PBB congeners strongly sorb to and clay, with log Koc values typically exceeding 6, resulting in minimal mobility and . Laboratory and field studies demonstrate negligible over extended periods; for instance, no significant breakdown of hexabromobiphenyl and heptabromobiphenyl isomers occurred after one year of under aerobic conditions. Estimated half-lives in exceed six months, often extending to years, as confirmed by dissipation trials showing persistence without measurable loss over six months. In aqueous environments, PBBs display low solubility (on the order of micrograms per liter for major congeners like FireMaster BP-6 components) and resist hydrolytic cleavage due to the stability of carbon-bromine bonds. Degradation in is minimal, with DT50 values greater than two months under conditions simulating natural or microbial activity. Photolysis occurs slowly in surface waters upon UV , primarily via stepwise debromination, but this pathway is insignificant in deeper or sediment-bound systems where light penetration is limited. microbial processes in sediments may facilitate partial debromination to lower-brominated congeners, though overall transformation rates remain slow, with no complete mineralization observed. Atmospheric persistence is governed by low and rapid scavenging; gas-phase PBBs react with hydroxyl radicals, yielding atmospheric half-lives of days to weeks for lower congeners, but deposition onto quickly dominates their fate. degradation by microorganisms is constrained by the absence of suitable catabolic pathways for highly brominated structures, though isolated reports indicate minor reductive debromination under conditions, producing penta- and tetrabromobiphenyls as metabolites. Overall, PBBs qualify as persistent organic pollutants, with environmental half-lives supporting long-range transport and accumulation in remote areas, as evidenced by their under frameworks assessing and bioaccumulative potential.

Bioaccumulation in Food Chains

Polybrominated biphenyls (PBBs) exhibit strong due to their high , low water solubility, and resistance to metabolic degradation, leading to storage primarily in adipose tissues, liver, and lipid-rich compartments across organisms. These properties facilitate , where concentrations increase at successive trophic levels in food chains, particularly in fatty tissues of higher predators. In aquatic systems, factors (BCFs) can reach 18,100 in like fathead minnows after 32 days of exposure, with even higher values (up to 1,440,000 on a lipid basis for certain congeners) observed in guppies. In the 1973 Michigan contamination incident, PBBs (primarily FireMaster FF-1) entered the agricultural through accidental mixing into livestock feed, affecting , , sheep, and . exposed to contaminated feed accumulated up to 1,000 mg/kg (1,000 ppm) in tissues, with concentrations reaching 2,000 ppb. from and showed residues up to 2,700 mg/kg and 4,600 mg/kg, respectively, prompting initial FDA tolerances of 1 ppm for and , later reduced to 0.3 ppm and further to 0.02 ppm by authorities. Plant uptake was minimal, with grass and root vegetables absorbing negligible amounts from contaminated soil (e.g., 35–1,260 μg/kg in farm soils), indicating that was predominantly animal-mediated via direct feed ingestion rather than soil-to-plant transfer. was evident in the transfer from feed to animal products, with residues persisting in manure-applied fields and secondarily in like fish-eating birds (0.02–0.25 mg/kg wet weight). Human exposure amplified this process through consumption of contaminated dairy, beef, and eggs, resulting in elevated PBB levels in (up to 419 ppb in residents, means of 603.9 ppb in workers) and (up to 6,000 ppb in chemical workers, 1,050 ppb in farmers). In nursing mothers, concentrations (median 250 μg/kg , up to 92,667 μg/kg) were 107–119 times higher than corresponding levels on a basis, demonstrating maternal-offspring transfer and further trophic magnification. This led to widespread contamination affecting millions, with slow elimination (half-lives of years) sustaining body burdens in exposed populations. Overall, PBB factors exceeded 1 across tested chains, underscoring risks to top consumers like humans reliant on animal-derived foods.

Michigan Contamination Incident

The 1973 Feed Mix-Up and Initial Spread

In , employees at the Michigan Chemical Corporation plant in , erroneously shipped bags of FireMaster BP-6—a () —to two Farm Bureau Services feed mills instead of NutriMaster, a cattle feed supplement. The mix-up stemmed from the similar appearance of the bags—both white with comparable labeling—and inadequate distinction in storage or shipping protocols, leading to an estimated 10 to 15 bags (approximately 500 to 1,000 pounds) of PBB being dispatched under the NutriMaster label. At the feed mills, the contaminated NutriMaster was blended into protein concentrate supplements at concentrations of about 0.25 (ppm), which were then distributed statewide to livestock farms, primarily for but also affecting , , and other animals. This initial dissemination reached hundreds of farms, with PBB rapidly transferring from feed to animal tissues, , eggs, and ; dairy cows, in particular, excreted PBB into milk fat at levels up to 100 times higher than in their feed due to . The contamination evaded detection for nearly a year, allowing widespread initial spread through the agricultural ; by late 1973, affected products had entered commercial markets, human diets, and even farm soils via , though symptoms in —such as reduced production and reproductive issues—began emerging in fall 1973 without immediate linkage to PBB. Confirmation of the feed mix-up occurred only in April 1974 after laboratory analysis of suspect samples, by which point an estimated 800 tons of contaminated feed had been produced and distributed.

Immediate Agricultural and Human Exposure

In 1973, polybrominated biphenyls (PBBs), marketed as FireMaster BP-6, were accidentally incorporated into livestock feed due to a labeling error at the Chemical Company, where bags of the were mistaken for supplement and shipped to a Farm Bureau Services feed mill. Approximately 10 to 20 fifty-pound bags, totaling 500 to 1,000 pounds of PBBs, were mixed into feed starting around May 2, 1973, leading to immediate ingestion by livestock on affected farms. Dairy cows exhibited rapid toxicity symptoms, including suppressed milk production, anorexia, , and reproductive failures such as abortions and stillbirths, with initial reports from as few as 25 farms documenting exposures up to 250 grams of PBB per animal head. The contamination spread beyond to , , sheep, and feeds, affecting over 500 farms by early 1974 and necessitating the quarantine and destruction of roughly 30,000 , 4,500 pigs, 1,500 sheep, 1.5 million chickens, and millions of eggs along with products. Human occurred primarily among farm operators and their families through direct handling of contaminated feed and consumption of tainted , , eggs, and other products from affected herds, with initial dissemination beginning in mid-1973 before widespread detection. residents, particularly on quarantined operations, showed elevated PBB levels—often 10 to 100 times higher than in the general —due to in and ongoing dietary intake. Acute symptoms reported by these groups included , , loss of , joint , , , skin rashes, and transient , though causation was confounded by and other factors; farmers consistently had the highest measurable exposures compared to other groups like chemical workers or non-farm residents. Prior to the April 1974 identification of PBB as the contaminant, an estimated 75% of total human via the had already occurred, as products from over 1,000 entered commercial distribution, potentially affecting millions of consumers through unpasteurized and processed .

Government Response and Quarantine Efforts

The Michigan Department of Agriculture () initiated quarantine measures shortly after animal symptoms emerged in late 1973, initially treating the issue as a potential infectious before confirming PBB contamination on April 24, 1974. Testing of , feed, and on suspected farms began promptly, with a of the contaminated Nutrimaster feed supplement issued on May 2, 1973, though full identification of PBB delayed comprehensive action. Quarantines restricted the sale, slaughter, or movement of animals and products from affected properties to contain spread through the . By early 1975, the had quarantined over 300 farms, expanding to more than 500 by program's end, with emphasis on operations due to PBB's lipophilic properties and transfer. Farms underwent ; those exceeding FDA action levels—initially 1 for (May 10, 1974) and (June 7, 1974), reduced to 0.3 by November 1974—faced mandatory depopulation. Contaminated herds were identified via tissue, , and fat analysis, prioritizing high-residue animals to minimize ongoing exposure. Destruction efforts resulted in the and disposal of approximately 30,000 , 4,500 , 1,500 sheep, and 1.5 million chickens, alongside over 800 tons of feed, 34,000 pounds of dried milk, 18,000 pounds of cheese, 2,500 pounds of butter, and 5 million eggs. Carcasses were buried or incinerated under state oversight to prevent environmental re-entry, with quarantined farms required to divert clean products or await clearance testing. support from the USDA's and Health Inspection Service (APHIS) included sampling over 2,900 heads from January 1975 to March 1977 to monitor for interstate risks. These measures, coordinated between state and federal agencies, successfully limited commercial distribution but imposed severe economic burdens on farmers, with compensation programs later addressing losses through state funds and lawsuits against responsible parties. continued post-quarantine to verify residue decline in surviving herds before release.

Health and Epidemiological Studies

Short-Term Observations from Exposed Populations

Following the 1973 contamination of livestock feed with polybrominated biphenyls (PBBs) in , exposed populations—primarily farm families handling contaminated animals and consumers of tainted , , and eggs—reported a range of acute symptoms within weeks to months of initial exposure. Common self-reported complaints included , , loss of appetite, and , observed in surveys of affected residents during 1973-1974. Dermatological effects were prominent, with increased prevalences of rashes, acne-like lesions (halogen acne), skin redness, peeling, scaling, itching, excessive sweating, and hair loss documented in clinical examinations of highly exposed individuals. Neurological and musculoskeletal symptoms were also frequently noted, such as headaches, , numbness or tingling in extremities, joint pain, and muscle aches, based on early epidemiological assessments by the Michigan Department of Public Health. Some reports included transient memory issues, though these were subjective and not consistently linked to PBB serum levels in initial evaluations. Objective clinical findings, however, showed limited correlations; physical exams and tests in the first year post-exposure revealed no clear dose-response relationships between PBB concentrations in blood or and abnormalities in , liver function, or other biomarkers. These short-term observations were derived from voluntary reporting and targeted screenings of over 3,000 Michigan residents with presumed high exposure, but confounding factors like stress from farm quarantines and economic losses complicated attribution. Early Agency for Toxic Substances and Disease Registry (ATSDR) reviews confirmed elevated subjective symptom rates but emphasized the absence of acute toxicity severe enough to require widespread hospitalization, consistent with PBB's low acute oral toxicity in humans (LD50 > 21 g/kg in animal models extrapolated to human data). Symptoms generally resolved without intervention, though persistent cutaneous changes were noted in a subset of cases with higher adipose PBB burdens (>1 ppm).

Long-Term Cohort Findings and Debates

A longitudinal of 3,954 individuals exposed to polybrominated biphenyls (PBBs) during the 1973 Michigan contamination incident, tracked from 1976 to 2021, revealed no significant association between serum PBB-153 levels and all-cause mortality, with hazard ratios (HR) of 1.03 (95% CI: 0.84–1.25) for females and 0.95 (95% CI: 0.80–1.13) for males. However, cancer-specific mortality showed sex-dependent patterns: higher PBB exposure correlated with elevated risk in females (HR 1.50, 95% CI: 1.02–2.22) but reduced risk in males (HR 0.68, 95% CI: 0.46–1.01), with () acting as an effect modifier in both sexes. No associations were observed for mortality. Additional analyses within this cohort have linked elevated PBB exposure to increased incidence among women, particularly at high exposure levels, as well as risks for digestive tract cancers. Reproductive health outcomes include disrupted function from in-utero exposure, higher prevalence, and broader impairments in exposed females. dysfunction, evidenced by altered hormone levels and function, has also been documented, alongside emerging links to autoimmune disorders and accelerated biological aging markers. The persistence of PBB-153, with a median of 13.5 years in women (ranging 8.1–27.2 years), contributes to protracted low-level exposures in this population. Debates surrounding these findings center on the inconsistent evidence for PBB carcinogenicity despite robust data demonstrating liver tumors and neoplastic nodules from . The sex-dimorphic cancer mortality patterns lack clear mechanistic explanations, with hypotheses including differential , hormonal interactions, or unmeasured confounders like factors and co-exposures to PCBs; the inverse male association has prompted speculation of protective effects or selection biases in the . While the U.S. National Toxicology Program classified PBBs in 2016 as "reasonably anticipated to be human carcinogens" based on sufficient animal evidence and limited human data, skeptics argue that cohort results show no overall mortality elevation and site-specific risks (e.g., ) may reflect rather than causation, necessitating larger, multi- validations to disentangle dose-response relationships and long-term epigenetic influences.

Intergenerational and Epigenetic Research

Research on intergenerational effects of polybrominated biphenyls (PBBs) primarily draws from the cohort exposed during the 1973-1974 contamination, tracking health outcomes in offspring of exposed parents. Studies have identified associations between parental PBB exposure and altered birth outcomes, including reduced birthweight linked to higher paternal serum PBB-153 levels, with each log-unit increase in paternal PBB corresponding to a 78-gram decrease in offspring birthweight after adjusting for confounders. Similarly, exposure via maternal PBB has been tied to developmental trajectories in male offspring, such as slower early growth followed by accelerated gains in body mass index and height by . These findings suggest potential transgenerational impacts, though causality remains inferred from observational data rather than direct mechanistic proof, with persistent PBB complicating isolation of prenatal versus postnatal effects. Reproductive and endocrine disruptions in subsequent generations have also been documented. Daughters of women exposed to PBB exhibited elevated progesterone levels and variations in characteristics, contrasting with some prior reports on direct exposure effects. Earlier maternal age at PBB exposure has correlated with shorter gestational lengths and lower birthweights in , independent of socioeconomic factors. Additionally, parental PBB levels have been associated with shifts in secondary ratios, with higher exposures linked to a slight female bias in , potentially reflecting endocrine disruption during or early embryogenesis. These epidemiological patterns, derived from longitudinal cohort data spanning decades, highlight risks but are limited by self-reported outcomes and the cohort's predominantly white, rural demographic, which may not generalize broadly. Epigenetic investigations, largely from animal models using FireMaster BP-6 (the commercial PBB mixture dominated by PBB-153), indicate mechanisms for heritable effects without genetic mutation. In male rats exposed developmentally to PBB-153, sperm exhibited disrupted patterns, including hypomethylation at imprinting control regions and altered expression of methyltransferase enzymes like DNMT3A and DNMT3B, leading to transgenerational defects in testicular development and . These changes persisted across F1 to F3 generations, suggesting epigenetic via gametic reprogramming failures. Human parallels are emerging, with PBB exposure associated with differential in blood near binding sites, potentially mediating and reproductive risks observed in the Michigan cohort. However, direct human epigenetic studies remain scarce, and while models provide causal insights, species differences in and exposure dosing warrant caution in extrapolation; no large-scale germline epigenome sequencing for PBB has confirmed multigenerational transmission.

Regulatory History and Alternatives

Domestic and International Bans

In the United States, the manufacture of polybrominated biphenyls (PBBs) was prohibited by the Environmental Protection Agency (EPA) in 1976 under the Toxic Substances Control Act (TSCA), following the 1973 agricultural contamination incident that exposed livestock feed to the chemical. Production had already been voluntarily discontinued by the sole domestic manufacturer, Michigan Chemical Corporation, in November 1974, after the incident revealed PBB's persistence and bioaccumulative properties. The EPA's ban extended to new uses, with existing stockpiles and wastes subject to strict disposal regulations to prevent further environmental release, though legacy contamination persists at sites like the Pine River area. Internationally, PBBs face restrictions rather than outright production bans in most jurisdictions, as commercial production was limited primarily to the U.S. and ceased globally by the late 1970s. In the , the Restriction of Hazardous Substances () Directive (2002/95/EC), effective July 1, 2006, prohibits PBBs in electrical and electronic equipment at concentrations exceeding 0.1% by weight in homogeneous materials, targeting their use as flame retardants in plastics and polymers to mitigate risks during and disposal. This measure aligns with broader efforts under the REACH Regulation to phase out persistent brominated compounds, though exemptions apply for certain legacy equipment until 2011 or later for specific applications. PBBs are not listed under the Stockholm Convention on Persistent Organic Pollutants, unlike related (PBDEs) added in 2009, reflecting their lower global production volume but shared concerns over toxicity and environmental persistence. Many countries, including those in and , have adopted similar product-specific restrictions via RoHS equivalents, effectively curtailing new imports and uses while addressing imported waste containing PBB residues.

Effectiveness of Regulations and Economic Impacts

Following the 1973 Michigan incident, the United States banned the manufacture and sale of polybrominated biphenyls (PBBs) in 1976, effectively halting domestic production by the late 1970s. This regulatory action, prompted by the accidental contamination of livestock feed, prevented further intentional introduction of PBBs into commerce, with no new manufacturing reported since. Internationally, PBBs, including hexabromobiphenyl, have been restricted under frameworks like the Stockholm Convention on Persistent Organic Pollutants, classifying them as endocrine disruptors and possible carcinogens due to their bioaccumulative nature. Despite these bans, regulations have proven only partially effective in eliminating exposure risks, as PBBs exhibit high environmental persistence and , leading to ongoing detection in , , and human tissues decades later. samples from former manufacturing sites in , analyzed years after the incident, retained PBB residues with altered congener profiles indicative of degradation resistance. Human biomonitoring shows that 80-85% of residents exposed in 1973-1974 maintain elevated serum PBB levels even after 40 years, reflecting slow elimination half-lives exceeding 10 years and intergenerational transfer via and . Remediation efforts, including farm quarantines and waste , reduced acute agricultural spread but failed to fully eradicate legacy contamination, necessitating continuous state-funded monitoring programs. The economic impacts of the PBB crisis were substantial, with direct costs from animal depopulation, product condemnation, and exceeding $215 million by 1979 estimates, encompassing lost , , and feed inventories. Over 500 farms were affected, involving the destruction of approximately 30,000 , 5,900 pigs, 1,470 sheep, and 1.5 million chickens, alongside 800 tons of contaminated feed and millions of units of and egg products. state expenditures for cleanup, testing, and compensation reached $75-100 million by 1987, including annual appropriations of at least $15 million for three years to trace and eliminate contaminated animals. Long-term costs include ongoing health studies and settlements, with the flame retardant industry's shift to alternatives adding indirect economic pressures, though no comprehensive post-ban industry-wide loss figures have been quantified. These impacts underscored the high financial burden of reactive , where prevention costs were dwarfed by crisis response but highlighted the value of preemptive bans in averting similar scales of damage elsewhere.

Development of Safer Substitutes

In the wake of the 1976 U.S. ban on PBB production, triggered by the 1973 Michigan contamination, manufacturers rapidly adopted (PBDEs) as direct substitutes in applications such as foams, high-impact polystyrene plastics, and textile coatings, where PBBs had previously provided additive flame suppression through vapor-phase radical scavenging. PBDE congeners like decaBDE, octaBDE, and pentaBDE formulations saw production escalate globally, reaching approximately 67,000 metric tons annually by 2001, enabling compliance with fire safety regulations like standards for electronics and furniture without immediate evidence of superior environmental safety. However, accumulating data on PBDE in wildlife and humans—evidenced by doubling concentrations in every five years from the 1970s to 2000s—revealed parallels to PBB persistence, spurring demands for non-brominated options. Regulatory pressures, including the European Union's 2004 restrictions on penta- and octaBDEs under the directive and U.S. voluntary phase-outs by major producers like Chemical in 2004, accelerated research into halogen-free flame retardants (HFFRs) starting in the late . -based additives, such as ammonium () and red , were developed for systems in polyolefins and epoxies, forming protective carbon char layers during combustion to insulate substrates and limit oxygen access, often synergized with like for enhanced efficacy at loadings of 20-30% by weight. These HFFRs avoided release, reducing smoke toxicity compared to brominated predecessors, and were commercialized for wire and circuit boards by firms like and by the early 2000s. Inorganic hydrated fillers, including aluminum trihydrate (ATH) and magnesium dihydroxide (MDH), also advanced as cost-effective alternatives for filled polymers like and , decomposing endothermically above 200°C to release that dilutes combustible gases and cools the material surface. Post-2000 innovations included nano-dispersions of these fillers to minimize mechanical property degradation at high loadings (up to 60%), enabling their use in flexible textiles and automotive interiors while meeting ASTM E84 flame spread criteria. Reactive incorporation of moieties directly into backbones, as in phosphonate-modified polyesters, further reduced migratability and environmental release, with prototypes tested for V-0 ratings in engineering resins by 2010. Despite these progresses, transitional substitutes like esters (e.g., tris(1,3-dichloro-2-propyl) phosphate or TDCPP) intended for foams post-pentaBDE often proved suboptimal, with studies indicating potential neurodevelopmental risks and endocrine disruption akin to legacy retardants, underscoring "regrettable substitution" patterns where efficacy prioritized over holistic hazard assessment. Ongoing EPA (DfE) alternatives assessments, initiated around 2010 for decaBDE, have prioritized multi-criteria evaluations favoring inherently less flammable materials like or redesign strategies over chemical additives, though adoption remains limited by performance trade-offs and regulatory inertia. By 2020, HFFR market share in electronics exceeded 50% in , driven by REACH evaluations confirming lower persistence profiles relative to brominated analogs.

Recent Developments and Ongoing Research

Advances in Detection and Remediation

Advances in detection of polybrominated biphenyls (PBBs) have primarily focused on enhancing sensitivity, selectivity, and efficiency in complex environmental and biological matrices. Gas chromatography coupled with mass spectrometry (GC-MS) remains the gold standard for PBB congener analysis, with recent refinements enabling quantification limits as low as parts per trillion in human serum and adipose tissue. For instance, a 2023 method quantified 11 PBB congeners alongside polychlorinated biphenyls using selective pressurized liquid extraction followed by GC-MS/MS, achieving recoveries exceeding 85% in serum samples. Liquid chromatography-inductively coupled plasma mass spectrometry (LC-ICP-MS) has emerged for polymer matrices, incorporating ultrasonic-assisted extraction to detect select PBBs at concentrations below 1 mg/kg, reducing sample preparation time by up to 50% compared to traditional Soxhlet methods. Solid-phase extraction (SPE) techniques have advanced through the use of mild post-modified metal-organic frameworks (MOFs), which provide high adsorption capacity for PBBs in water and soil extracts, minimizing matrix interferences and enabling detection via GC-MS with limits of detection around 0.1 ng/L. These MOF-based SPE methods, developed around 2024, emphasize by avoiding harsh solvents and achieving extraction efficiencies over 95% for hexa- and heptabromo congeners. In biological , such as post-Michigan studies, PBB levels are tracked using evolved extraction protocols that transitioned from basic solvent methods in the to automated SPE-GC-MS by the , allowing longitudinal detection of decay rates with half-lives estimated at 10-20 years for major congeners like PBB-153. Remediation of PBB-contaminated sites remains challenging due to the compounds' high persistence, low water solubility, and bioaccumulative nature, with half-lives in exceeding decades under aerobic conditions. Traditional approaches prioritize physical removal, such as excavation and at temperatures above 1000°C to achieve >99.99% destruction efficiency, as applied in limited post-1970s cleanups where contaminated soils were landfilled or thermally treated. No large-scale in situ biodegradation methods have been commercialized for PBBs, though laboratory studies indicate slow microbial debromination under conditions, with potential enhancements via using dehalogenating , but field efficacy remains unproven as of 2025. Emerging research explores , leveraging plants like ryegrass to uptake PBBs from , with translocation factors up to 0.5 observed in trials, though scaling is limited by slow kinetics and plant toxicity thresholds. The U.S. EPA notes ongoing lab-scale investigations into , such as with UV/TiO2 systems, which degrade PBBs by 70-90% in spiked matrices over hours, but application requires further optimization to penetrate sorbed fractions. Overall, remediation strategies emphasize and monitored natural attenuation where exposure risks are low, given the absence of validated destructive technologies for widespread environmental media as of recent assessments.

Current Risk Assessments from Persistent Exposures

Persistent exposures to polybrominated biphenyls (PBBs) occur primarily through legacy contamination in soil, sediments, and the , given their high environmental persistence and , with half-lives in humans estimated at 10–20 years or longer, leading to in fatty tissues. In the general U.S. , levels are low, typically around 0.026 ng/ median for key congeners, reflecting diminished production since the 1970s bans, though detectable residues persist in , fish, and near contaminated sites like those from the 1973 incident. Highly exposed cohorts, such as farm families, exhibit elevated levels (up to 236 ng/ range), sustaining risks from dietary intake of contaminated , , and . Health risk assessments emphasize potential carcinogenicity, with the International Agency for Research on Cancer (IARC) classifying PBBs as Group 2A ("probably carcinogenic to humans") based on sufficient animal evidence of liver, , and mammary tumors, despite limited human epidemiological data showing no clear overall cancer excess in exposed Michigan residents. A 2025 long-term cohort analysis of over 3,000 Michigan participants found sex-specific associations between higher PBB exposure and increased cancer mortality risk, particularly among females for and endocrine-related cancers, attributing this to endocrine-disrupting properties like hormone interference observed in studies at doses of 0.3–10 mg/kg/day. However, factors such as lifestyle and co-exposures limit causal attribution, and broader population studies report no definitive links to non-cancer outcomes like neurodevelopmental deficits beyond acute high-dose scenarios. Emerging research highlights non-cancer risks from chronic low-level exposures, including accelerated biological aging; a 2019 study of residents linked higher PBB serum levels to shortened length, a marker of , independent of age and , suggesting oxidative stress mechanisms akin to those in animal models. Transformation products from may amplify , with 2025 assessments indicating elevated carcinogenic and developmental risks for certain PBB byproducts, though human relevance remains understudied due to analytical challenges in detection. Regulatory bodies like the U.S. EPA and ATSDR deem current general population risks low, prioritizing remediation at hotspots, but recommend ongoing for vulnerable groups like lactating women, given transgenerational transfer via and . Overall, while supports precautionary measures against , definitive causal risks from ambient exposures require further longitudinal data to disentangle from historical peaks.

References

  1. [1]
    Polybrominated Biphenyls (PBBs) | Public Health Statement - CDC
    Polybrominated biphenyls (PBBs) are chemicals that were added to plastics used in a variety of consumer products, such as computer monitors, televisions, ...
  2. [2]
    Polybrominated Biphenyl - an overview | ScienceDirect Topics
    Polybrominated biphenyls (PBBs) are a class of brominated hydrocarbons consisting of a central biphenyl structure to which 1–10 bromine atoms are attached.
  3. [3]
    [PDF] Technical Fact Sheet – Polybrominated Biphenyls (PBBs) - US EPA
    ❖ PBBs were formerly used as additive flame retardants in synthetic fibers and molded plastics. They are no longer used in the United. States (ATSDR 2004; NTP ...
  4. [4]
    Polybrominated Biphenyls (PBBs) | ToxFAQs™ | ATSDR - CDC
    Polybrominated biphenyls (PBBs) are chemicals produced by human activity and are found in plastics used in many consumer products to make them difficult to burn ...
  5. [5]
    Fate of polybrominated biphenyls (PBB's) in soils. Persistence and ...
    Fate of polybrominated biphenyls (PBB's) in soils. Persistence and plant uptake. Click to copy article linkArticle link copied! Lee W. Jacobs · Sheng-Fu ...<|separator|>
  6. [6]
    Poly-brominated Biphenyls (PBBs) in Michigan
    Polybrominated biphenyls (PBBs) are man-made chemicals commonly used as a fire retardant in plastic products before they were banned in the United States in ...
  7. [7]
    The History of PBBs in Michigan
    PBBs were in the food chain through contaminated milk and dairy products, beef, pork, lamb, chicken, and eggs.
  8. [8]
    Michigan's PBB contamination: 50 years later
    May 10, 2023 · In 1973, toxic flame retardant was mistakenly sent to Michigan farmers as livestock feed, causing an environmental health crisis. To this day, ...
  9. [9]
    The PBB episode in Michigan: an overall appraisal - PubMed
    Cattle on about 25 Michigan farms were exposed to as much as 250 g per head of PBB when it was accidentally mixed in cattle feed in 1973 to 1974.
  10. [10]
    [PDF] Michigan PBB Contamination
    The PBB was mixed into livestock feed and consumed by cattle, pigs, and chickens. Contaminated farm products were distributed throughout the state until the ...
  11. [11]
    Health Effects from PBB Exposure - State of Michigan
    Some Michigan residents who were exposed to polybrominated biphenyls (PBBs) in the 1973-74 contamination incident complained of nausea, abdominal pain, loss of ...
  12. [12]
    Pediatric aspects of the Michigan polybrominated biphenyl ...
    In 1973, Michigan cattle feed was accidentally contaminated with polybrominated biphenyls (PBB) and subsequently PBB has been found in the serum, body fat, ...
  13. [13]
    Environmental politics and science: the case of PBB contamination ...
    This article examines how politics and science interacted against a background of uncertainty to shape policy in the case of environmental contamination.
  14. [14]
    CHEMICAL AND PHYSICAL INFORMATION - NCBI - NIH
    PBBs are a class of structurally similar brominated hydrocarbons in which 2–10 bromine atoms are attached to the biphenyl molecule.
  15. [15]
    [PDF] POLYCHLORINATED BIPHENYLS AND POLYBROMINATED ...
    Feb 19, 2013 · PBBs have a molecular formula of. C12H(10-n-m)Br(n+m) where n + m = 1 to 10, i.e. from monobromobiphenyl to decabromobiphenyl. There are 209 ...<|separator|>
  16. [16]
    Pbb 153 | C12H4Br6 | CID 42948 - PubChem - NIH
    2,2',4,4',5,5'-Hexabromobiphenyl is a polybrominated biphenyl. Polybrominated biphenyls (PBBs) are a group of 209 synthetic organic compounds with 1-10 ...
  17. [17]
    [PDF] Toxicological Profile for Polybrominated Biphenyls
    The chemical identities of hexabromobiphenyl, octabromobiphenyl, decabromobiphenyl (BB 209), and BB 153, the most abundant congener in commercial FireMaster FF ...
  18. [18]
    [DOC] Hexabromobiphenyl - Stockholm Convention
    The hexabromo congeners exist as 42 possible isomeric forms, which are listed with CAS and IUPAC numbers in US ATSDR (2004) and in document INF 2.
  19. [19]
    [PDF] TECHNICAL FACT SHEET – PBDEs and PBBs - US EPA
    ❖ PBBs also exist as mixtures of congeners. They were produced as three primary homologs: hexabromobiphenyl (hexaBB), octabromobiphenyl (octaBB) and ...
  20. [20]
    Polybrominated biphenyls (PBBs) (HSG 83, 1993) - INCHEM
    The halogenated biphenyls have been categorized on the basis of structure. Category 1 comprises isomers and congeners lacking orthosubstituents (coplanar PBBs).
  21. [21]
    Table 4-3, Physical and Chemical Properties of Selected PBBsa
    Color, White, White ; Physical state, Solid, Solid ; Melting point, 72 °C · 200–250 °C; 365–367 °C (for industrial product) ; Boiling point, No data, No data ...
  22. [22]
    POLYBROMINATED BIPHENYL - NOAA - CAMEO Chemicals
    Alternate Chemical Names · BROMINATED BIPHENYL · FIREMASTER FF-1 · 2,4,5,2',4',5'-HEXABROMOBIPHENYL · PBB · POLYBROMINATED BIPHENYL · POLYBROMINATED BIPHENYL (FF-1).
  23. [23]
    [PDF] RoC Profile: Polybrominated Biphenyls - National Toxicology Program
    Log Kow. 6.39. Water solubility. 0.011 mg/L at 25°C. Vapor pressure. 5.2 × 10–8 mm Hg at 25°C. Source: ChemIDplus 2010. Use. PBBs are no longer used in the ...
  24. [24]
    Polybrominated biphenyls (EHC 152, 1994) - INCHEM
    1.2 Manufacturing processes The process of manufacturing PBBs consists of a Friedel-Crafts type reaction in which biphenyl is reacted with bromine in the ...
  25. [25]
    PRODUCTION, IMPORT/EXPORT, USE, AND DISPOSAL - NCBI - NIH
    The commercial production of polybrominated biphenyls (PBBs) generally involves bromination of biphenyl, a process involving a much more specific reaction and ...
  26. [26]
    [PDF] Toxicological Profile for Polybrominated Biphenyls
    Physical and Chemical Properties. Many of the relevant physical and chemical properties of the. PBBs are not available (see Table 4-3). More data on the ...<|separator|>
  27. [27]
    [PDF] ATSDR Polybrominated Biphenyls (PBBs) Tox Profile
    Sep 1, 2004 · Polybrominated biphenyls (PBBs) are brominated organic compounds used as flame retardant additives in plastics, textiles, and other materials.
  28. [28]
    [PDF] Toxicological Profile for Polybrominated Biphenyls
    Polybrominated biphenyls (PBBs) are brominated organic compounds used as flame retardant additives in plastics, textiles, and other materials.
  29. [29]
    Polybrominated Biphenyl - an overview | ScienceDirect Topics
    PBBs induce cytochrome P450 isozymes from the CYP1A family. The ability to induce CYP1A enzymes is related to the binding affinity of congeners to the Ah ...
  30. [30]
    HEALTH EFFECTS - Toxicological Profile for Polybrominated ... - NCBI
    Several studies examined the acute dermal effects of commercial PBB mixtures in rabbits. ... PBB congeners also studied structure-toxicity relationships.
  31. [31]
    Human developmental toxicity mechanism of polybrominated ...
    Jun 1, 2022 · Polybrominated biphenyls (PBBs) can bioaccumulate in nature and are toxic to humans. Long-time exposure to PBBs in pregnant women can lead ...
  32. [32]
    Intergenerational effects of endocrine-disrupting compounds - NIH
    Jun 11, 2018 · This review will focus on the potential mechanisms through which PBB exposure may lead to intergenerational health risks.
  33. [33]
    Carcinogenicity of polyhalogenated biphenyls: PCBs and PBBs
    This review discusses the role of PCBs and PBBs in the process of carcinogenesis. The mutagenicity/genotoxicity of these compounds, as well as their initiation/ ...
  34. [34]
    Assessment of the Hazards of Polybrominated Biphenyls - epa nepis
    HEW-FDA Division of Chemical Technology. Polybrom- inated biphenyls in fish, milk, eggs and cheese. Summary report of polybrominated biphenyl findings. 11/10/74 ...
  35. [35]
    Polybrominated Biphenyls - 15th Report on Carcinogens - NCBI
    Polybrominated biphenyls (PBBs) are reasonably anticipated to be human carcinogens based on sufficient evidence of carcinogenicity from studies in experimental ...
  36. [36]
    Polychlorinated Biphenyls and Polybrominated ... - IARC Publications
    Polychlorinated biphenyls are a class of aromatic compounds comprising 209 congeners, each containing 1 to 10 chlorine atoms attached to a biphenyl nucleus.
  37. [37]
    Mortality of individuals in a long-term cohort exposed to ...
    Jul 1, 2025 · Environmental exposure to polybrominated biphenyl (PBB) associates with an increased rate of biological aging. Aging (Albany NY). 2019;11(15): ...
  38. [38]
    Field concentrations and persistence of polybrominated biphenyls in ...
    No evidence of significant degradation of PBB was noted after 1 year incubation in soil. When 14C hexabromobiphenyl and heptabromobiphenyl isomers were ...
  39. [39]
    [PDF] United Nations Environment Programme - Stockholm Convention
    Nov 21, 2006 · The EHC review (1994), concludes that polybrominated biphenyls are stable and persistent in the environment. The degradation of PBBs by purely ...<|separator|>
  40. [40]
    [PDF] A Mixed Bag in Michigan: The PBB Story - tephinet
    In September 1973, a farmer in southwest. Michigan noticed a decline in the feed consumption and milk production of his 400 dairy.
  41. [41]
    A State of the Science Review of Human Health Effects of the ...
    Review. 28 April 2025. A State of the Science Review of Human Health Effects of the Michigan Polybrominated Biphenyl Contamination after Five Decades. Authors ...<|control11|><|separator|>
  42. [42]
  43. [43]
    [PDF] Federal Efforts to Protect Consumers from Polybrominated Biphenyl ...
    Jun 8, 1977 · Throughout the program. Michigan identified and quarantined contaminated herds. APHIS initiated sampling of cattle and swine for PBB in January ...
  44. [44]
    [PDF] PBBs (Polybrominated Biphenyls) in Michigan
    Polybrominated biphenyls (PBBs) are man-made chemicals that were used as fire retardants in plastics that were used in a variety of consumer products. PBB is a ...
  45. [45]
    [PDF] The Pbb Episode in Michigan: An Overall Appraisal
    Exposure of cattle to feed that contained over 1000 ppm PBB unquestionably produced adverse production and health effects. 23 However, many clinical signs that ...<|separator|>
  46. [46]
    The Case of PBB Contamination in Michigan
    levels of PBB could damage animal and human health, helped redefine the PBB problem as a public health hazard to. Michigan consumers. Many group members also ...
  47. [47]
    Cutaneous effects of exposure to polybrominated biphenyls (PBBs)
    In 1973 an environmental accident occurred in northern Michigan in which 1000–2000 pounds of the toxic fire retardant polybrominated biphenyl (PBB) was ...
  48. [48]
    RELEVANCE TO PUBLIC HEALTH - Toxicological Profile for ... - NCBI
    Polybrominated biphenyls (PBBs) are brominated organic compounds used as flame retardant additives in plastics, textiles, and other materials.
  49. [49]
    The human health effects of exposure to polybrominated biphenyls
    There was no relationship between PBB levels and physical or laboratory abnormalities. Present evidence suggests that people exposed to PBB have few objective ...Missing: short- | Show results with:short-
  50. [50]
    [PDF] Toxicological Profile for Polybrominated Biphenyls
    PBB toxicity is not known. It has been suggested, however, that the mechanism for some congeners is related to the enhancement of gene expression triggered ...
  51. [51]
    Mortality of individuals in a long-term cohort exposed to ...
    Jul 1, 2025 · PBB and other endocrine disruptors can affect multiple organ systems, impacting normal hormone processes in the body. Endocrine-related effects ...
  52. [52]
  53. [53]
    PBB Research - State of Michigan
    There is some evidence linking high amounts of PBB exposure in the Michigan Long-Term Study to an increased risk of cancers of the breast and the digestive ...
  54. [54]
  55. [55]
  56. [56]
  57. [57]
  58. [58]
    Environmental exposure to polybrominated biphenyl (PBB ...
    The association between PBB and increased age acceleration could be caused by many biological mechanisms that cannot be directly tested in this study.Missing: bioaccumulation | Show results with:bioaccumulation
  59. [59]
    Elimination of PBB-153; findings from a cohort of Michigan adults
    Mar 1, 2023 · Overall, PBB exposure has been linked to negative acute and chronic outcomes in several organ systems (Curtis et al., 2018) including but not ...
  60. [60]
    Critical Reviews in: Carcinogenicity of Polyhalogenated Biphenyls
    Long-term exposure to PCBs and PBBs in animals has been associated with the induction of neoplastic nodules in the liver and in some cases hepatocellular ...
  61. [61]
    Birth Outcomes Associated with Paternal Polybrominated and ...
    The findings suggest that increased paternal PBB and PCB levels negatively impact offspring birthweight, and paternal PCB levels may negatively impact ...
  62. [62]
    Birth outcomes associated with paternal polybrominated and ...
    The findings suggest that increased paternal PBB and PCB levels negatively impact offspring birthweight, and paternal PCB levels may negatively impact ...
  63. [63]
    In utero exposure to a brominated flame retardant and male growth ...
    The current study investigates associations between in utero PBB exposure and growth and developmental outcomes among the male offspring of cohort members.
  64. [64]
    In-utero exposure to polybrominated biphenyl (PBB) and menstrual ...
    Research based on the Michigan PBB registry has shown associations between exposure to PBB and many reproductive health outcomes (Small et al., 2011). In ...
  65. [65]
    The influence of age at exposure to PBBs on birth outcomes
    This study examined the association between early age at exposure to polybrominated biphenyls (PBBs) and subsequent birth weight and gestational length in ...
  66. [66]
    A cohort study of the association between secondary sex ratio and ...
    Aug 15, 2009 · This cohort presents a unique opportunity to study the association between parental exposures to PBB and offspring sex ratio.
  67. [67]
    Detrimental effects of flame retardant, PBB153, exposure on sperm ...
    May 22, 2020 · PBB153 exposure alters the epigenome by disrupting methyltransferase activity leading to defects in imprint establishment causing altered gene expression.<|control11|><|separator|>
  68. [68]
    Detrimental effects of flame retardant, PBB153, exposure on sperm ...
    May 22, 2020 · PBB153 exposure alters the epigenome by disrupting methyltransferase activity leading to defects in imprint establishment causing altered gene expression.
  69. [69]
    Research links fire retardant exposure to hormone-related DNA ...
    Feb 4, 2019 · We found that those with higher levels of exposure to PBB have common patterns of DNA methylation that are similar to what we see in hormones ...Missing: offspring studies
  70. [70]
    RoHS Directive - Environment - European Commission
    It currently restricts the use of ten substances: lead, cadmium, mercury, hexavalent chromium, polybrominated biphenyls (PBB) and polybrominated diphenyl ethers ...
  71. [71]
    Environment: EU ban on hazardous substances in electrical and ...
    Four heavy metals and two groups of brominated flame retardants are being banned as they can pose a direct risk to workers' health when waste electrical and ...
  72. [72]
    New EU Limits on PBDEs Set to Reshape Chemicals and Recycling ...
    Jul 31, 2025 · New EU limits on PBDEs, toxic flame retardants, are effective from July 24, 2025, with limits decreasing to 350 mg/kg by 2025 and 200 mg/kg by ...Missing: PBB | Show results with:PBB
  73. [73]
    Polybrominated Diphenyl Ethers, Polybrominated Biphenyls, and ...
    Due to concerns about increasing exposure and potential associations with adverse health outcomes, the US government banned the production of PBBs in 1976 and ...
  74. [74]
    Tracing the toxic legacy of PBB contamination | Emory University
    In 1973, bags of a fire-retardant chemical called PBB, polybrominated biphenyl, were accidently mixed into livestock feed and sold to farmers throughout the ...<|separator|>
  75. [75]
    [PDF] The Michigan PBB Incident - CDC Stacks
    As expected, PBB was discovered in beef --and also in milk and milk products ... Polybrominated biphenyls (PBB) environmental contamination in Michigan, 1973-1976 ...
  76. [76]
    [PDF] After 40 years, effect of Michigan's PBB crisis still not fully known
    Feb 9, 2015 · Some 1.5 million chickens, 30,000 cattle, 5,900 pigs and 1,470 sheep consumed the feed and became contaminated with PBBs. More than 500 farms ...<|separator|>
  77. [77]
    [PDF] Polybrominated Diphenyl Ethers (PBDEs) Action Plan I ... - EPA
    Dec 30, 2009 · Serum concentrations of poly6brominated diphenyl ethers (PBDEs) and polybrominated biphenyl (PBB) in the United States population: 2003-2004.
  78. [78]
    Flame retardants and their risks - Chicago Tribune
    May 10, 2012 · History Production of PBDEs increased rapidly as PBBs were phased out of use. Chemical companies stopped making penta and octa after Europe ...
  79. [79]
    Brominated Flame Retardants To Be Voluntarily Phased Out - EPA
    Nov 3, 2003 · “The company's decision to swiftly cease production of these chemicals by 2004 will accelerate the shift to safer alternatives. ... fire safety ...
  80. [80]
    Recent developments in the chemistry of halogen-free flame ...
    An overview of recent developments of the chemistry of halogen-free flame retardant polymers is presented in this paper.
  81. [81]
    A Review of the Development History of halogen-free flame retardants
    The development history of halogen-free flame retardants is closely related to the promotion of environmental protection regulations and technological ...<|separator|>
  82. [82]
    Organophosphate Ester Flame Retardants: Are They a Regrettable ...
    Oct 21, 2019 · In this paper, we ask whether OPFRs are a better choice than PBDEs. To address this question, we compared OPFRs with PBDEs for a wide range of properties.<|separator|>
  83. [83]
    [PDF] Report on Alternatives to the Flame Retardant DecaBDE
    Polybrominated diphenyl ether. (PBDE)-induced alterations in vitamin A and thyroid hormone concentrations in the rat during lactation and early postnatal ...
  84. [84]
    New flame retardants and applications trend towards PIN ... - Pinfa
    Feb 12, 2025 · PIN flame retardants do not release halogen acids, and so can achieve CTI performance around twice as high as halogenated FRs. Budenheim's ...
  85. [85]
    Quantification of Polybrominated and Polychlorinated Biphenyls in ...
    We have developed a highly sensitive and selective analytical method capable of quantifying a total of 15 polybrominated and polychlorinated biphenyls (11 PBBs ...<|separator|>
  86. [86]
    Determination of Selected Polybrominated Diphenylethers and ...
    Aug 6, 2025 · A new method has been developed for the determination of selected polybrominated diphenylethers (PBDEs) and polybrominated biphenyl (PBB) in ...
  87. [87]
    Mild post-modified metal–organic frameworks applied as solid ...
    This study established a simple and green sample pretreatment method for the detection of PBBs in complex matrices.
  88. [88]
    Determinants of Polybrominated Biphenyl Serum Decay among ...
    The PBB detection methods used in this study have changed over time. During the years 1976–1978, the extraction method was used to detect PBBs. From 1978 to ...Missing: advances | Show results with:advances
  89. [89]
    [PDF] Technical Fact Sheet - Polybrominated Diphenyl Ethers (PBDEs ...
    ❖ PBDEs exist as mixtures of distinct chemicals called congeners with unique molecular structures. The PBDE congeners may differ in the total number or position ...<|separator|>
  90. [90]
    Mortality of individuals in a long-term cohort exposed to ...
    This study is a long-term follow-up of individuals exposed to polybrominated biphenyls (PBBs). Widespread contamination of PBBs began in 1973 in Michigan ...
  91. [91]
    Mortality of individuals in a long-term cohort exposed to ... - PubMed
    Jul 1, 2025 · This comprehensive study found that the association between PBB exposure and cancer mortality risk varied by sex.Missing: outcomes | Show results with:outcomes
  92. [92]
    Extrapolation of PBBs Environmental Transformation Mechanisms ...
    Feb 19, 2025 · Results showed that the transformation products of PBBs and their substitutes exhibit high toxicity risks (i.e., potential carcinogenicity, ...